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Moreover, the binding of anaphylatoxins (C3a and C5a) to C5L2 only increases the immune cell degranulation potential upon cross-linking high-affinity immunoglobulin E (IgE) receptor by a pertussis toxin-sensitive mechanism
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Nanodrug modulates premetastatic niche and suppresses metastatic lung adenocarcinoma via programmed cell death ligand 1 blockade and STING pathway activation
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