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Furthermore, microbiome-mediated inflammation may accelerate BE progression via lipopolysaccharide (LPS)-mediated activation of Toll-like receptor 4 (TLR-4), which leads to IL-18 production and pro-inflammatory cascades [36]

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Polyneuropathy typically presents gradually, beginning with paresthesia known as “burning feet,” followed by gait disturbances and variable muscle weakness in the lower limbs ( In the CNS, thiamine deficiency can lead to Wernicke’s encephalopathy, characterized by nystagmus, ophthalmoplegia, mental status changes, and unsteady stance and gait, due to apoptotic cell death in sensitive brain areas, mainly areas with high metabolic requirement, causing symmetrically distributed lesions among structures such as the third and fourth ventricles, besides dorsomedial thalamus (42)