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These findings indicate that the inhibition of the inflammatory environment and ECM remodeling process was promoted by using CD5/ β LNP-FAPCAR 2.2
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DG reduces portal pressure by restoring SOD3 activity in the portal area, counteracting oxidative stress in portal vein macrophages, and maintaining the bioavailability of nitric oxide and prostacyclin (PGI 2 ) (Zhao et al., 2022)
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