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The interactions between shared molecular mediators of BCAA and lipid metabolism [e.g., mammalian target of rapamycin complex 1 (mTORC1), AMP-activated protein kinase, peroxisome proliferator-activated receptor (PPAR)-γ coactivator-1α (PGC-1α), and PPARα] can also be a contributing factor to the relationship between BCAA and lipid metabolism in the liver (28–32)

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doi: 10.1016/j.addr.2005.07.009