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These findings indicate that HSP inhibition is unlikely to function as a broadly effective, histology‐independent strategy and may instead require tumors with defined dependence on particular HSP isoforms, clients, or co‐chaperone complexes

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Recent research has demonstrated that trans-3-indoleacrylic acid, a tryptophan metabolite derived from the gut microbiota, suppresses ferroptosis and facilitates colorectal cancer progression [94]