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2 Pathological mechanisms of pulmonary fibrosis: the crosstalk between cellular metabolic dysregulation and oxidative stress The onset of pulmonary fibrosis is typically triggered by chronic or recurrent injury to lung tissue, with damage to alveolar epithelial cells, particularly type II alveolar epithelial cells (AT2 cells), recognized as a critical initiating event
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Despite the possibility of such reactions, allergy shots are generally well-tolerated, especially when patients adhere to a regular dosing schedule and do not miss doses
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