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demonstrate that NIX-mediated mitophagy functions as a novel effector pathway of oncogenic KRAS, promotes the elimination of functional mitochondrial content to stimulate cell proliferation and augments redox homeostasis by increasing glycolytic metabolism and augmenting redox robustness [53]

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or parasitic mites) by about 2%, 5%, 10%, 20%, 30%, 40%, 50%, 60%, 70%, 80%, 90%, 100%, or greater than 100% relative to a reference level (e.g., a level found in a pest that does not receive a PMP composition)