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Interestingly, murine studies have shown that intestinal overexpression of ACE2 is associated with increased abundance of anti-inflammatory microbial species, in contrast to ACE2 knockout models, which exhibit more proinflammatory profiles [86]
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salivarius and Pediococcus ssp., e18, amnion) were associated with activation and balance function of the innate and adaptive immune systems (lactic acid bacteria) and attenuation of processes related to the development of the immune system and dysregulation of immunological mechanisms (Enterobacteriaceae) at hatch (170)
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